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Its like there are factors in this virus that are not yet understood at all.
I think this is definitely true. Can different strains have highly differing levels of mortality?
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There is a RNA tracking map of it, there are a few sub varieties atm.
Maybe they also tried to compared mortality for these but it is super hard to do atm, see also badboybjorns post above.
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@t-v It wouldn't surprise me.
I can't believe whatever mild symptoms I and my flatmate had were the same strain that hospitalised a fit guy in his 30's from my workshop building and killed my uncle.
Either there are different strains or there is a genetic element to who is most vulnerable (disregarding age and pre-existing conditions).
I'm lucky in that I've only had flu twice in my 40 years. Could this be related to the mild level of symptoms I got for what I assume was covid19?
Who knows..
JWestland
nomoreknees
No thats what concerns me (well not only for sweden but for the world). It would have been much better if the virus had a large spread everywhere all rdy (obviously since lower death rate) but also like u say, that makes u wonder about our 30% with antibodies in stockholm since this was a projection from a rather small group of testing in comparison to the spain survey for instance. But they have been in complete lockdown since late march or similar? So i still have some hope that the stockholm numbers can hold somewhat true when they release the next test results after the weekend.
There was also that german study of village that found 15%? had had it and only a mortality rate of about .3% or similar which makes one wonder how it can differ so much. Are the tests they used in spain not reliable perhaps? not picking up on every case?
Theres also places like india that leaves me somewhat puzzled.. how can the slum of the big cities in india not produce massive deaths way way higher than beeing reported.
Its like there are factors in this virus that are not yet understood at all. There is otherwise no logic or way to little accurate data at least.