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Did I miss a meeting? Surely VO2max is pretty relevant to a GC contender.
I think the implication was that focusing exclusively on the effect of Salbutamol on V02 Max on someone who already has an exceptionally high scores is not relevant and you should instead be testing how how it might improve other physiological responses.
Saying that salbutamol doesn't increase your V02 Max is not the same as saying it's not performance enhancing. Quick google-fu turned up this which also indicates that V02 capacity being the primary limitation of performance in elite athletes might be inaccurate, seems fairly well argued.
http://www.scienceofrunning.com/2009/12/fallacy-of-vo2max-and-vo2max.html
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I think the implication was that focusing exclusively on the effect of Salbutamol on V02 Max on someone who already has an exceptionally high scores is not relevant and you should instead be testing how how it might improve other physiological responses
Studies have looked for other responses, and found nothing when using the dosing regime permitted by WADA. Science being what it is, we can't say that there's nothing to find if you looked in the right places using the right test subjects, but it's all irrelevant anyway under current regulations, because you don't need a TUE for 1600μg/day inhaled, so the playing field is level regardless of whether there is some as yet undiscovered benefit.
gbj_tester
Depends what you're trying to prove. Most of the studies I've seen are testing the performance enhancing effect of therapeutic doses of inhaled drugs, to determine whether giving TUEs to asthmatics has unintended consequences. Testing large systemic doses (e.g. 4-8mg orally) is a different matter, but that kind of regime would be an out of competition thing, you'd have to be unbelievably stupid to keep it up in the racing season.
Did I miss a meeting? Surely VO2max is pretty relevant to a GC contender.